Articles Related to Glucose
Overweight and Obesity and their Relationship with Glucose Dysregulation in the Nigerian Youth
Fasting plasma glucose is a risk factor for diabetes and cardiovascular disease in obese children. This study aimed to evaluate the
association between WC, BMI and WHtR and blood glucose in children.
Effects of Ramadan Fasting on Anthropometric Measures, Blood Pressure and Glucose Level among Type 2 Diabetic Patients on Metformin Treatment
Fasting from dawn to dusk during Ramadan is obligatory for all healthy adult Muslims.
Metabolic Effects of an Inositol-Resveratrol Nutraceutical Combination in Non-Diabetic Overweight/Obese Subjects with Altered Glucose Tolerance
Insulin resistance (IR) is almost constistently associated with overweight or obesity in both diabetic and non-diabetic subjects.
Nutraceutical compounds improving insulin resistance (IR) may be useful in prevention and treatment of cardiovascular (CV) risk factors in metabolic diseases.
Safety Concerns of Glaucoma Chemotherapy among G6PD Deficient Glaucoma Patients: A Pilot Study
The aim of this study was to assess the potential acute adverse effects associated with the use of anti-glaucoma medications
among glaucoma patients with Glucose-6-Phosphate Dehydrogenase deficiency.
How Blood Glucose Relevance with Falooda Ice Cream Loving?
The amount of glucose or sugar that is present in our blood is termed as blood glucose, normal value of it’s should be 5.4 or 5.6 mmol/ L in humans. Mainly two types of blood glucose are discussed hyperglycemia high amount of sugar and hypoglycemia low amount of sugar. Falooda ice cream is a sweet and delicious so it also contains too much sugar that is necessary for our health and maintaing blood glucose level but by eating too much it can cause hyperglycemia. Hyperglycemia may damage eyes, blood vessels and kidneys. There is significant relation between falooda ice cream loving and blood glucose.
Computer Modeling of Convective Mass Transfer of Glucose, Oxygen and Carbon Dioxide in the Neurovascular Unit
Glucose and oxygen are obligatory energy substrates for the brain and their continuous supply to the neurons, as well as the elimination of the end product – carbon dioxide, is of paramount importance for brain physiology and survival. Transport of these substrates from the brain capillaries to the neurons presents a challenging issue.
High Glucose Modulates Responsiveness to Estrogens of Human Derived Female Cultured Osteoblasts and in Osteoblastic Cell Lines
Human female- derived osteoblast- like cells in culture (hObs) and the cell lines (SaOS2 and hFOB) express mRNAs involved in bone biology and physiology such as estrogen receptor α (ERα) and α (ERα), vitamin D receptor (VDR), 1α, 25 (OH) vitamin D3 hydroxylase (1OHase) and 12 and 15 lipoxygenases (12LO and 15LO). These mRNAs are modulated by estrogenic compounds. Since the skeletal protective effects of estrogens are not discernible in diabetic women, we tested the estrogenic modulations of these parameters in cells grown in growth medium containing high glucose (HG; 9.0g/L; 44mM) compared to normal glucose (NG; 4.5g/L; 22mM). HG significantly increased DNA synthesis (DNA) and creatine kinase specific activity (CK). Stimulations of DNA but not of CK by estradiol-17β (E2), by 4, 4’, 4’’-[4-propyl-(1H)-pyrazol-1, 3, 5- triyl] tris-phenol (PPT; ERa specific agonist), or by 2, 3-bis (4-hydroxyphenyl)-propionitrile (DPN; ER β specific agonist), were modulated by HG. HG Itself up regulated the expression of mRNA of 12LO and 15LO and to less extent ERβ and VDR, but had no effect on ERα and 1OHase mRNA expression. The different hormonal treatments modulated the expressions of VDR, 1OHase, 12LO and 15LO mRNAs which were reduced in HG, whereas the induction of their products 1α, 25 dihydroxy-vitamin D3 (1,25D) and 12- and 15- hydroxyeicosatetraenoic acid (12 and 15 HETE) were only slightly affected by HG. The exact mechanism of HG effects on bone cell responses and its relationship to human bone physiology is not yet clear.
microRNA 181a-5p Reprogramed Glucose and Lipid Metabolism in Non- Small Cell Lung Cancer
microRNAs play critical roles in cancer metabolisms. miRNA-181a-5p is significantly down-regulated in non-small lung cancer tissue and mesenchymal like lung cancer cells. Epithelial-mesenchymal transition mechanisms and cancer metabolism are controlled by the same signaling pathway.
Metabolic Syndrome in Indigenous Amerindian Women in Suriname; Less on Waist and More on Weight?
The indigenous Amerindian populations living in the southern interior part of Suriname have to date largely maintained their traditional hunter-gatherer life-style. In this study we compared the prevalence of metabolic syndrome (MeTS) and its component risk factors between indigenous Amerindian women of the interior of Suriname, with indigenous Amerindian women living in the coastal-rural areas who have a more urbanized lifestyle. We focused on women since the Suriname Health study showed that Indigenous women had the second highest MeTS prevalence nationwide.
Blonanserin N-Oxide Lowers Glucose Levels in Animal Models
The N-oxide of anti-psychotic drug Blonanserin (BLNO) was synthesized and tested as a novel molecule to understand its ability in
reducing blood glucose levels.
Epidemiologic Characteristics of Diabetes in an Affected South Texas Population
Our research study on diabetic patients in a community in South Texas have shown that increasing age, male gender, married status, smoking history, limited education, and birth in Mexico are risk factors for observed increasing HbA1c and FBG. Elevated lipoprotein levels are particularly observed in type 2 diabetes. Low insulin levels and high BMI in this high risk population correlate to the severity of their diabetes. Moreover we have observed that being born in Mexico influences the patient’s method of treatment with use of oral or combined oral and parenteral routes with poor control of their diabetes. Perhaps the Hispanics born in Mexico may have limited knowledge of their disease influenced by their educational level, socioeconomic status, and prior misconceptions on appropriate methods of treatment(s).
Glycemic Index and Glycemic Load of a Carbohydrate-Rich and Protein-Rich Formula Diet
Obesity is well associated in various chronic diseases such as type 2 diabetes, hyperinsulinemia, dyslipidemia and atherosclerosis. Thus, for persons with obesity and diabetes, food with low GI are recommended in order to lower the glycemic response.
Glucose Testing and Lifestyle Counseling of Obese and Overweight Patients: A Survey of Primary Care Physicians in the United States
In the United States (U.S.), the costs for health care and lost productivity due to diagnosed diabetes has increased from $174 billion in 2007 to $245 billion in 2012, making it one of the fastest growing public health burdens. Nearly 26 million adults have diabetes, and another 79 million have prediabetes, a condition that increases the risk for developing type 2 diabetes. Clearly to reduce the costs and improve health, preventing diabetes must be a priority.
Predictors of Obstructive Sleep Apnea Risk among Blacks with Metabolic Syndrome
Identification of risk factors for obstructive sleep apnea (OSA) is important to enable comprehensive intervention to reduce OSA-related cardiovascular disease (CVD). The metabolic syndrome outcome study (MetSO) provides a unique opportunity to address these factors. This study investigated risk of OSA among blacks with metabolic syndrome.
Pharmacokinetics, Biotransformation, Distribution and Excretion of Empagliflozin, a Sodium-Glucose Co-Transporter (SGLT 2) Inhibitor, in Mice, Rats, and Dogs
The metabolism, pharmacokinetics, excretion and distribution of a sodium-glucose co-transporter (SGLT 2) inhibitor, empagliflozin, were studied in mice, rats and beagle dogs following a single oral or intravenous administration of [14C]-empagliflozin. Empagliflozin was well absorbed in all species after oral administration.
Editorial Board Members Related to Glucose
ASISH K. SAHA
Associate Professor
Department of Medicine
Boston University
United States
Department of Medicine
Boston University
United States
Andrey Budanov
Assistant Professor
Department of Human and Molecular Genetics
Massey Cancer Center
United States
Department of Human and Molecular Genetics
Massey Cancer Center
United States
Jason W. Locasale
Assistant Professor
Division of Nutritional Sciences
Cornell University
United States
Division of Nutritional Sciences
Cornell University
United States
Rodrigo Fernandez Valdivia
Assistant Professor
Department of Pathology
Wayne State University School of Medicine
United States
Department of Pathology
Wayne State University School of Medicine
United States
SABIRA KHATUN
Professor
Faculty of Electrical and Electronics Engineering
University Malaysia Pahang
Malaysia
Faculty of Electrical and Electronics Engineering
University Malaysia Pahang
Malaysia