Articles Related to acetylcholinesterase
A Novel Neuroprotective and Antioxidative Efficacy of a Unique Combination of Standardized Huperzia serrata, Convolvulus pluricaulis and Celastrus paniculatus Extracts
Physical and environmental stress in conjunction with hectic lifestyle and unhealthy food habits are the major cause of diverse neurodegenerative disorders. Oxidative neuronal injury and acetylcholine deficiency have a major impact on learning and memory retention. Most of the treatment strategies are based on the improvement of cholinergic function in the brain and one of the emerging therapeutic targets is to enhance the acetylcholine level in the brain. Standardized botanical extracts including Huperzia serrata (1% Huperzine A, CogniUp), Convolvulus pluricaulis (SP) and Celastrus paniculatus (JY) have been demonstrated to attenuate brain function by serving as a natural acetylcholinesterase (AChE) inhibitor.
Impact of the Biofield Energy Treated Proprietary Novel Formulation on Memory and Cognition Function in Neuronal Cell
The current study was investigated for the in vitro cognitive effect of the Consciousness Energy Healing (The Trivedi Effect®) Treated novel proprietary test formulation in terms of acetylcholinesterase (AChE) inhibition, neuroprotection, and neurite outgrowth assays in two different neuronal cell-lines (SHS-Y5Y and PC-12 Cells). The test formulation divided into two parts; one part was received the Biofield Energy Treatment by Mr. Mahendra Kumar Trivedi and termed as the Biofield Energy Treated sample, while the other part was denoted as the untreated test sample.
Beneficial Effects of Vitamin E against Indomethacin-induced Glutathione Depletion, Acetylcholinesterase Activity, (Na+,K+)-ATPase Activity and Osmotic Fragility of Erythrocyte
Indomethacin, is a well-known non-steroidal anti-inflammatory drug (NSAID), a cyclooxygenase inhibitor and extensively used as a medicine to reduce pain, fever, stiffness, swelling or inflammatory responses. Indomethacin inhibits both COX-1 and COX-2, which in turn, inhibits the production of prostaglandins in the gastrointestinal tract and disrupts the mucosal lining in the gastrointestinal tract leading to intestinal bleeding and perforation.
Protective Ability of Vitamin E Against Acetylsalicylic Acid-Induced Glutathione Depletion, Acetylcholinesterase and (Na+ ,K+ )-ATPase Activities, and Erythrocyte Osmotic Fragility
Acetylsalicylic acid (ASA), a well-recognized non-steroidal anti-inflammatory drug (NSAID), is well known for the treatment of fever, pain alleviation and inflammatory conditions, especially for the prevention of cardiovascular complications and disorders. However, ASA has been reported to cause hepato- and gastro-toxicities.
Expression Profile of Hepatic Metallothionein-I and ATP7B, and Brain Metallothionein-III and Acetyl cholinesterase Genes in Wistar Rat Model for Non-Wilsonian Brain Copper Toxicosis
Cognitive waning due to chronic copper (Cu) intoxication in animal models is increasingly being reported; notwithstanding, information regarding molecular basis of Cu accumulation and neurobehavioral impairments remains fragmentary.