Open Access Journal

Journal of Neurology and Neurological Disorders

ISSN: 2454-4981 IF: 3.6* DOI: 10.15744

Email: jnnd@annexpublishers.com

Submit Manuscript → Browse Articles Instructions for Authors

Journal at a Glance

Review typeDouble-blind

Target decision~21 days

Submission feeNone

LicenceCC BY 4.0

Word limitsNone

About

About the Journal

Journal of Neurology and Neurological Disorders (JNND) is a multidisciplinary refereed journal which publishes significant, innovative research works that extends the standards of excellence established by the scientific academicians. JNND accepts articles in all areas of Neurology, developments in clinical neuroscience, current diagnosis, prevention methods and treatment of neurological disorders, etc.

Why publish with us

Built for researchers

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Fast peer review

Target first decision within 21 days. Double-blind review by 2-3 field experts.

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Fully open access

All articles CC BY 4.0. No paywalls. Compliant with funder mandates worldwide.

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DOI & indexing

Every article receives a unique DOI. Indexed in PubMed, Google Scholar, CrossRef.

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Rapid publication

Published within 7 days of acceptance. No unnecessary delays in dissemination.

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Global & equitable

Readers in 170+ countries. Waiver program available for authors from developing nations.

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No content limits

No word limits. Multiple article formats: research, reviews, case reports, short communications.

How it works

Simple. Transparent. Streamlined.

Submit

Upload your manuscript via our online portal. Quality check within 48 hours.

Review

Double-blind peer review by 2-3 field experts. Target decision: ~21 days.

Revise

Constructive feedback from reviewers. Dedicated editorial support throughout.

Publish

Published within 7 days. DOI assigned via CrossRef. Globally indexed.

Submit Manuscript →

Highlights

Research Highlights

Psychological Factors and Conditioned Pain Modulation: A Meta-Analysis.

The aim of this meta-analysis was to analyze the findings regarding the associations between the CPM responses and psychological factors in both healthy subjects and pain patients.

Novel plasminogen gene mutations in Turkish patients with type I plasminogen deficiency.

In this study, five individual Turkish patients and nine Turkish families with type 1 Plg deficiency were investigated for PLG gene mutations. All of the coding regions of the PLG gene mutations were screened for mutations using denaturing high-pressure liquid chromatography (DHPLC).

ATM, MacroH2A.1, and SASP: The Checks and Balances of Cellular Senescence.

Oncogene activation is usually not enough to induce cancer, but causes cells to arrest proliferation, alter chromatin structure, and increase protein secretion. In this issue of Molecular Cell, Chen et al. (2015) implicate the histone variant macroH2A.1 in the regulation of senescence.

Neuropathology and Cellular Pathogenesis of Spinocerebellar Ataxia Type 12.

SCA12 is a progressive autosomal-dominant disorder, caused by a CAG/CTG repeat expansion in PPP2R2B on chromosome 5q32, and characterized by tremor, gait ataxia, hyperreflexia, dysmetria, abnormal eye movements, anxiety, depression, and sometimes cognitive impairment.